Aluminum Facts
Jeff Lioon, jr of Oligoscan has performed over 6500 scans and states "The most common metal that I see in people, across ALL ages is unequivocally Aluminum."
HTMA Experts agree with Jeff and state up to 93% of people tested show elevated aluminum levels in their hair, making it the most frequently detected toxic metal in clinical hair analysis.
Aluminum is the most abundant metal and the third most abundant element in the Earth’s crust, after oxygen and silicon, at about 8% by weight.
99% of it is bound to silicon in the form of aluminosilicate minerals.
Due to its widespread use in Stratospheric Aerosol Injection aka geoengineering, anti-perspirants, cookware, food dyes (aluminum lake), anti-caking agents, vaccine adjuvants, metallurgy, and rare earth mineral extraction background environmental exposure will continue to increase over time.
Christopher Exley, the most accomplished aluminum researcher, knew this and used high silica water (ie Fiji) to deplete aluminum from people with neurodegenerative disorders. He was able to double the excretion of aluminum in Alzheimer's patients and get 158% increase in aluminum excretion from those with Multiple Sclerosis.
Exley also showed up to a 7mg sweat excretion of aluminum with sauna.
95% of aluminum is filtered by the kidneys with no direct liver involvement.
Hydration and electrolytes, particularly potassium, is critical for supporting the kidneys in filtering aluminum.
The ATDSR states a healthy adult has 30-50mg of total body burden of aluminum.
Additionally, ATDSR states 50% is stored in bone, 25% in the lungs, and the remaining 25% spread across other tissues (brain).
Unbound aluminum forms complexes with citrate, which can cross the BBB independently of transferrin.
Aluminum binds to amyloid-β and tau proteins, forming stable complexes that resist clearance. These aggregates are hallmarks of Alzheimer’s disease and act as "sinks" for aluminum.
A Frontiers in Neurology review on aluminum notes that aluminum binds ATP 103 times higher relative to its usual co-factor Mg(II), emphasizing just how strongly aluminum can out‑compete magnesium where it gains access.
Al3+ blocks mitochondrial electron transport chain complexes in mammals, broadly inhibiting complexes I–V and ATP synthase via oxidative and binding effects. As a result, aluminum toxicity is a significant factor in mitochondrial dysfunction and decreased cellular energy.
Al disrupts osteoblast activity and collagen cross-linking, causing osteomalacia (soft bones)
Aluminum also suppresses parathyroid hormone (PTH), impairing calcium regulation and worsening bone demineralization.
The Intervertebral Disc Study showed strong correlations between high aluminum and low silicon levels.
Aluminum exposure redirects collagen-based silicon toward detoxification, creating a functional deficiency in collagen tissues. This weakens connective tissues, increasing susceptibility to injuries (e.g., tendon tears, joint degeneration).